Tissue adhesive vs suture
It is very uncertain whether tissue adhesive changes wound infection compared with sutures (very low certainty).
- RR 1.72 [0.92, 3.16]10 studies · 744 participantsVery low
It is very uncertain whether tissue adhesive changes wound infection compared with sutures (very low certainty).
Tissue adhesive showed no clear difference in wound dehiscence compared with adhesive tape (low certainty).
Tissue adhesive showed no clear difference in wound infection compared with adhesive tape (low certainty).
Tissue adhesive showed no clear difference in wound dehiscence compared with staples (low certainty).
It is very uncertain whether tissue adhesive changes wound infection compared with staples (very low certainty).
Tissue adhesive showed no clear difference in wound dehiscence compared with other methods (low certainty).
Tissue adhesive showed no clear difference in wound infection compared with other methods (low certainty).
It is very uncertain whether high viscosity tissue adhesive changes wound dehiscence compared with low viscosity tissue adhesive (very low certainty).
It is very uncertain whether anterior changes non‐fusion at compared with posterior surgical approaches (very low certainty).
It is very uncertain whether anterior changes complications Number of events compared with posterior surgical approaches (very low certainty).
Non‐steroidal antiandrogen monotherapy probably leads to higher overall survival compared with LHRH agonists or surgical castration monotherapy (moderate certainty).
Non‐steroidal antiandrogen monotherapy probably leads to higher clinical progression compared with LHRH agonists or surgical castration monotherapy (moderate certainty).
Non‐steroidal antiandrogen monotherapy probably leads to higher treatment failure compared with LHRH agonists or surgical castration monotherapy (moderate certainty).
Non‐steroidal antiandrogen monotherapy probably leads to higher breast pain compared with LHRH agonists or surgical castration monotherapy (moderate certainty).
Non‐steroidal antiandrogen monotherapy probably leads to higher gynaecomastia compared with LHRH agonists or surgical castration monotherapy (moderate certainty).
Non‐steroidal antiandrogen monotherapy probably leads to lower hot flashes compared with LHRH agonists or surgical castration monotherapy (moderate certainty).